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PLoS ONE 18:e0283163

doi: 10.1186/s40364-024-00620-x 325

Additionally, in young animals, escape latency was further reduced in PXN HIGH compared to CAF HIGH by approximately 14% ( P = 0.01)

For example, NAPQI, the toxic metabolite, is primarily the product of CYP2E1 metabolism, and there is evidence that younger children have a relative immaturity in CYP2E1 metabolism, which would substantially reduce the production of this toxic metabolite in infants.7 After birth, CYP2E1 protein expression and activity increases, reaching adult levels by about 1 year of age.7 The ability of the liver to metabolize acetaminophen changes with age owing to differences in activity of these key metabolic pathways